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Hey everyone,

Just letting you guys know that I'm doing okay, nothing really serious is happening.  I've been just a bit distracted on doing the things that I want to do.  Jobs are getting worse and worse, I don't see anything getting better for anyone and that includes me.  I've just been on low key for the remainder of the spring witht he exception of this weekend.  It's going to be hot!!!

Usually I would have more things to jot down on this blog than I do on my myspace pages.  I'm even far back from putting wrestling captions up on my Hardy fan page.  I guess it's just laziness or just plain boring.  Today I'm just going to redo my nails, sign the lease, and send my resume out again, see how good my luck is..right now I'm hungry but I don't know what to eat.

Sorry if I'm rambling and make no sense, but anyone who wakes up at 5am wakes up to pee and eat.lol...But right now I'm late to watch my golden girls.  Yeah even at 27 I live comfortable, My husband doesnt mind me being at home, but I think for his sake and mine, the way how this economy is going, we won't be able to afford rent...

So let see what happens, will write a blog soon

Ni

A contest for Wrestling Fans

A few weeks ago, On Wrestlemania 25 weekend, My Husband and I made up a contest for wrestling fans who have a youtube account, an award for Best Fan Promo Video.  It was entitled Hardy v Hardy WM XXV.  The person who one goes by the youtube ID chigatorsfan23.  He recieved his award just a few days ago and is happy.  The reason why I'm writing this, is because, if there are any women who have youtube accounts and love wrestling, you could be picked by me or my husband.  The way how this contest works is by us looking at a certain Wrestling Story Line.  Will look on youtube for Fan Made Promo videos on Youtube for that story line and you'll receive a message from either myself or my husband whom is known as Gilligan.  We post a message to 10 candidates that they have been choosen with all the rules and regulations.

The First award was on April 5, 2009, which was short notice, but the next Award will be announced on May 1, which is next week.  So if you know anyone who loves the WWE and have Home Made Promos, let them know because at the beginning of next week, we will choose 10 people, notify them on may 1 and the winner will be announced during the Extreme Rules PPV in June.

If you need further clarity email us at dabronxhardyzpage@yahoo.com or on www.myspace.com/dabronxhardyzpage

Hope to hear from you soon

Ni

Questions on Living arrangements

Why is it easy for a man to move in with a woman?  Why is it easy for a woman to move in with a man?  What is marriage?  What is common-law?  Is it the same?  Are there any rule difference?

I am living with my husband..and there were some problems that his son and daughter-in-law were going through.  So, her family doesn't get involved becuz they know how there daughter is.  See I'm a young step-mom, my step son is 3 years older than me.  Well to get to the point, My husband's daughter-in-law and son are having problems in there marriage..and ofcourse my bf is always involved and I try not to be, but this event took pace about 3 days ago.  His daughter in law calls up stating to my husband that his son (whew), wants to fight her cousin all because she had cheated on him b4 they got married and after, so in othe words, he doesn't trust her.  So he thought the guy wasn't her cousin and all hell broke loose, so my husband insisted that I go with him, but again, I try not to get involved.  So we went to there apartment complex, knocked on there door and who opens the door, His Son!!!... My husband was stressed out, wanted to stab the poor guy, and he's yelling at his son saying "You got me like this all stressed the F8ck out!" so he was going to get in and he stated "I'm gonna F8ck him up" so the daughter in law who cried wolf, went into his face after calling him and bitching, cursed him out. 

Now I dont know about you, but I have my standards on this as I'm sure you will agree or disagree, but my parents are alive, and I've never allowed anyone curse at my parents, especially in front of me, so his son just allowed it and stopped his dad from what we thought "the marriage was over" fight.  SO I was pissed, I couldnt lay a hand on her out of respect for my step-son, but I'm puerto rican and mexican, I'd be damned to have that person come visit me in my home and chill like nothing happened.. "No Bitch you just fucking cursed my man out, and he didn't want you to in his house and wanted nothing to do with you" 

So now it's April 17 and she apologized but to him not me plus I have standards, I have parents (remind you), that was a dis-respect to me, if she talked to my husband like that imagine her parents.  She tried to talk to me but I didn't want to, she tried to make conversation, I paid her no mind, I have my standards, I'm sure you have yours. 

Being that I don't pay rent, and the apartment is not under my name nor am I on the lease, I can't make any decisions in this apartment, one of them was, "She's not crossing the door"  "She's not welcomed here"  "She's nothing but a drama queen, I'm to old for this shit kinda thing."  Does anyone get my point?

Everytime they come over, I'll head over to my mom's, reason, because of the respect I have for my step-son, and being raised in a hispanic family with boundaries and rules, Women like her need to be shot in the mouth for starting trouble, playing teen games, and acting like a degenerate.  And she already knows I don't like her, before I never said it, but now, after ignoring her and not wanting to do with her, I dont like her whatso freakin ever. 

This sucks for me but hey, I moved in and I can't make the rules, so from now on everytime they fight and my husband wants to get in, he's on his own, I got my own thing to do and my own shit to worry about.  Hypocrit is the only word that comes to mind.  His house his rules.  I'm just going to stay quiet until things get shakin, I'll hit you up at another date.

Twin Bartter's Syndrome

Bartter's Syndrome is a rare kidney disorder among premature infants.  It has been studied by few nephrologists in the United States.  I know a set of kids that deal with their renal disorder since birth.  While many of you think that there is a cure for the syndrome, there isn't, but there is medication that can help.  Pending on how severe Bartter's Syndrome is, it rates from 1-5, 1 being the lesser of the syndrome but still fatal and 5 being the most severe and watched over from medical providers to families who deal with it every day.  I am one of those family members who deals with seeing younger siblngs who were born with Bartter's.  Visiting the Hosptial has been more Home for my younger siblings than there own.  As you will read, you will learn about the different types of Bartter's, plus you'll read a true life story about my younger siblings and how they deal with it...especially if your 6 years old.  I was planning on writing something that may draw attention..I hope this is one of them..Twin Bartter's syndrome is about a set of twins (boy) and (girl) who were born on August 2, 2002 from there biological Mother with Bartter's Syndrome at 26 weeks (5 1/2 months) and other complications.  They were adopted in October 19, 2005 from my Family.  The details are real but the names will be Kept confidential, so if you can't read on, I would understand, but it will make you double take and look at your children and see how lucky you and they really are.

Bartter’s Syndrome

Bartter’s Syndrome is an inherited defect in the renal tubules that causes low potassium levels, low chloride levels, which in turn causes metabolic alkalosis. Bartter Syndrome, is not a single disorder but rather a set of closely related disorders. These Bartter-like syndromes share many of the same physiologic derangements, but differ with regard to the age of onset, the presenting symptoms, the magnitude of urinary potassium (K) and prostaglandin excretion, and the extent of urinary calcium excretion.

At least three clinical phenotypes have been distinguished:

• Classic Bartter Syndrome;
• The Gitelman Variant;
• The Antenatal Variant (also termed Hyperprostaglandin E Syndrome).

There are also many patients who have other variant that they believe may be variants of Bartter’s that has not yet been identified. Whatever type of electrolyte problems you are having will determine what type of Bartter’s syndrome you have. There are many patients who have characteristics of Classic Bartter’s and Gitelman’s combined. From the research I have done it appears that many of the patients that have Classic Bartter’s initially seem to change as they get older and start having problems with magnesium which is more characteristic of Gitelman’s. This is one thing that has baffled many nephrologist’s and researchers. These patients fall into that category of having both or many of them have their diagnoses switched from Classic Bartter’s to Gitelman’s. No one knows why this occurs.

Classic Bartter Syndrome

Bartter’s Syndrome was first discovered in 1962 by Frederic Bartter. Bartter described this syndrome in two African-American patients: a 5 year old boy and a 25 year old man with a long history of slow growth, weakness and fatigue. On high sodium diets, both patients had normal blood pressure and high urinary aldosterone excretion, resulting in metabolic alkalosis. This syndrome is a disorder of the renal tubules in the kidneys that causes a higher than normal Aldosterone level and low serum potassium levels which lead to metabolic alkalosis. Patients with this syndrome share a myriad of clinical symptoms and laboratory abnormalities. Most notable are:

• Profound hypokalemia (Very low serum potassium levels)
• Increased urinary excretion of potassium (K) and prostaglandins.
• Normal blood pressure despite elevated plasma renin and aldosterone levels. (High renin and aldosterone causes hypertension in a normal healthy person. In Bartter’s the Renin and Aldosterone are elevated but the patients do not have hypertension)
• Hypochloremic (Low serum chloride) metabolic alkalosis
• A relative vascular resistance to the pressor effects of exogenous angiotensin II
• Hyperplasia of the juxtaglomerular apparatus

Bartter described this combination of juxtaglomerular hyperplasia, hyperaldosteronism and hypokalemic alkalosis in two African-American subjects: a 5yr old boy and a 25yr old man with a long history of slow growth, weakness and fatigue. On high sodium diets, both subjects had normal blood pressure and high urinary aldosterone excretion, resulting in hyperbicarbonatemia.

Initially, it was considered a vascular disease. In the 1970’s, when prostaglandins were discovered, it was found that Bartter’s Syndrome patients overproduced prostaglandins. If treated with a prostaglandin inhibitor, aldosterone levels returned to normal. Plasma potassium levels did not. Subsequently, experimental potassium deficiency induced prostaglandin production and many of the symptoms of Bartter’s Syndrome. The suggested problem was not intravascular, but a renal tubular problem.
Frequency In the US

There are both familial (Inherited) and sporadic forms of Bartter’s and Gitelman’s syndromes. The prevalence of this disorder is not precisely known, but one study cites an estimate of 1.2 per million. Although many cases appear to be sporadic, Bartter’s syndrome is well described in siblings as well as in children of consanguineous marriages. This pattern of transmission suggests an autosomal recessive* mode of inheritance. However, in a few kindreds, the inheritance pattern is more consistent with autosomal dominant** transmission.

*Autosomal Recessive - Both parents carry the gene which is passed on to the child.

**Autosomal Dominant - One parent carries the gene which is passed on to the child.
Frequency Internationally

Estimates of incidences vary from country to country.

In Costa Rica, incidence of neonatal Bartter’s from live births is estimated at 1.2/100,000, higher if all preterm births are considered. No evidence of consanguinity was found in the Costa Rican cohort.

In Kuwait, the incidence of consanguineous marriages or related families in Bartter’s syndrome patients is higher than 50% and the incidence is 1.7/100,000.

In Sweden, the incidence has been calculated as 1.2/1,000,000. Of the twenty-eight patients reported by Rudin, seven cases came from three families. The others were unrelated.

Sex

Bartter’s syndrome is inherited as an autosomal recessive trait, there is no gender preference.

Race

There is no racial predisposition for Bartter’s Syndrome.

Age

Classic Bartter’s syndrome is usually diagnosed in childhood or adolescence.

Symptoms

• Fatigue
• Polyuria (Increased urination)
• Polydipsia (Increased Thirst)
• Nocturia (Waking up at night to urinate)
• Generalized weakness
• Salt Cravings
• Dehydration
• Mental confusion
• Vomiting
• Muscle weakness
• Muscle spasms
• Tetany
• Failure to thrive
• Short stature (If untreated)

Lab and Physical findings

• Low serum potassium levels
• Low-normal magnesium levels
• Increased renin
• Increased aldosterone
• Metabolic Alkalosis
• Increased Prostaglandin E2 excretion
• Normal-high urinary calcium excretion
• Normal-high urinary Mg excretion
• Normal-low serum Mg levels (20% have decreased Mg levels)
• Normal - Low Blood Pressure
• Increased urinary potassium excretion
• Increased plasma angiotensin II
• Nephrocalcinosis
• Tetany, muscle spasms, Chvostek’s sign and Trousseau’s sign may be seen in hypokalemia, hypocalcemia, and hypomagnesemia patients. In the older literature rickets was also occasionally reported.
• In 1997, Madrigal described a type of this syndrome in Costa Rica in sixteen of the twenty patients with a “peculiar facies, distinguished by a triangularly shaped face, large eyes, and protruding ears”.
• Another eight had sensorineural hearing loss determined by audiography.

In addition to these biochemical perturbations, a small subset of patients have developed progressive renal insufficiency due to severe tubulo-interstitial nephritis. It is not clear whether the loss of renal function in these patients was a direct consequence of their primary molecular defect or secondary phenomenon related to their chronic hypokalemia.
What Causes this Syndrome?

The most accepted explanation for Classic Bartter Syndrome involves a primary defect in Cl transport in the TAL. This is a part of one of the tubules in the kidneys where electrolytes are absorbed into the bloodstream.
Treatments

There is no cure for Bartter’s syndrome. The treatments consist of supplements to replace what is lost and medications to prevent urinary wasting of potassium and magnesium. In younger children growth hormone may be used to prevent the short stature and prostaglandin inhibitors to decrease the elevated prostaglandin levels.

• Potassium Chloride Supplements
• Magnesium Supplements
• Spironolactone
• Amilioride
• Triamterene
• Indomethacin
• Captopril
• Growth Hormone

Prognosis

The limited prognostic information available suggests that early diagnosis and appropriate treatment of infants and young children with Classic Bartter Syndrome may improve growth and perhaps neurointellectual development. On the other hand, sustained hypokalemia and hyperreninemia can cause progressive tubulointerstitial nephritis, resulting in end-stage-renal disease (Kidney failure). With early treatment of the electrolyte imbalances the prognosis for patients with Classic Bartter Syndrome is good.